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Inflammation occurs by both molecular and cellular components on the surface of blood microvascular endothelial cells.1,7,8 Pathological features in ischemic stroke, such as cell necrosis, debris produced by dead cells, and reactive oxygen species (ROS), may induce inflammation in nerve cells through activation of microglia and astrocytes, as well as infiltration of leukocytes from peripheral blood circulation.9,10 The pathophysiology of ...

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